BioMEMS and Biomedical Nanotechnology: Volume IV: by Rashid Bashir, Mauro Ferrari, Steven Wereley

By Rashid Bashir, Mauro Ferrari, Steven Wereley

Volume four of the multi-volume reference, BioMEMS and Biomedical Nanotechnology В is a balanced evaluate of key facets of BioMEMS sensors, together with (i) BioMEMS sensors and fabrics, (ii) technique of manipulating organic entities on the microscale, and (iii) micro-fluidics and characterization. those 3 sections offer a succinct evaluation of vital themes inside of a unmarried volume.

This quantity is particularly good illustrated with a number of the figuresВ in color.

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Psychiatric syndromes Huntington in his original paper recognized that mental deterioration was one of the characteristic features of the disorder that was to bear his name; he noted ‘the tendency to insanity, and sometimes that form of insanity that leads to suicide’. Most of the subsequent case reports, however, concentrated on the chorea rather than the psychiatric manifestations of HD. One of the more systematic of the early studies on mental disorder in HD was conducted by Phelps (1892) at Rochester, Minnesota.

1908c) Dr Charles Rollin Gorman. I. Personal sketch. II. His relation to the chorea question. Neurographs 1:144–147. —— (1908d) Irving Whitehall Lyon MD. I. Personal sketch. II. Location of his cases. Neurographs 1:147–149. Bruyn GW (1968) Huntington’s chorea. Historical, clinical and laboratory synopsis. In: Handbook of Neurology (eds PJ Vinken and GW Bruyn) Vol. 16, pp. 298–378. Amsterdam: Elsevier. —— Baro F and Myrianthopoulos NC (1974) A centennial bibliography of Huntington’s chorea 1872–1972.

These findings were confirmed by later workers (Alzheimer 1911; Pfeiffer 1913), but it was not until the 1920s that there was general agreement that the brain changes in HD were primarily degenerative and atrophic, and that the caudate nucleus was preferentially involved in the process. Neuropathological studies have recently again become of central importance in HD research, first with the advent of detailed quantitative analyses, and now with the recognition of neuronal inclusions as a key element in the pathology of both human HD and in the transgenic mouse models, as discussed in later sections of the book.

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